Tony Charman
Chair of clinical child psychology
King's College London
From this contributor
Building an autism research registry: Q&A with Tony Charman
A purpose-built database of participants who have shared genomic and behavioral data could give clinical trials a boost, Charman says.
Building an autism research registry: Q&A with Tony Charman
Your questions about the Lancet Commission and ‘profound autism,’ answered
Tony Charman and Catherine Lord answer questions from Spectrum’s webinar on the Lancet Commission’s recommendations for autism research.
Your questions about the Lancet Commission and ‘profound autism,’ answered
Separate thinking skills underlie autism, attention deficit
Theory of mind difficulties are likely to be more central to autism than to attention deficit hyperactive disorder, whereas executive function problems are more often associated with the latter.
Separate thinking skills underlie autism, attention deficit
Tony Charman: Longitudinal studies for autism research
Clinicians and autism researchers should learn the early signs of autism and take into account an individual’s developmental trajectory, says Tony Charman.
Tony Charman: Longitudinal studies for autism research
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The chimeric mice could help untangle the biology of human brain development or conditions such as autism.
What if Hegel were a neuroscientist?
Andrea Gambarotto shares what Hegel and enactivism have in common: an agency-first approach to understanding minds and brains.
What if Hegel were a neuroscientist?
Andrea Gambarotto shares what Hegel and enactivism have in common: an agency-first approach to understanding minds and brains.
This paper changed my life: Transformative work in addiction neuroscience
A 2011 Nature study found that loss of a nicotinic receptor subunit leads to increased nicotine intake in mice. The work set the standard for how to functionally validate genetic association study findings in neuroscience.
This paper changed my life: Transformative work in addiction neuroscience
A 2011 Nature study found that loss of a nicotinic receptor subunit leads to increased nicotine intake in mice. The work set the standard for how to functionally validate genetic association study findings in neuroscience.