Sven Sandin is associate professor of psychiatry and a member of the Seaver Autism Center for Research and Treatment at the Icahn School of Medicine at Mount Sinai and a statistician in the medical epidemiology and biostatistics department at the Karolinska Institutet. He has more than 20 years of experience with epidemiological studies, primarily using the Swedish and Nordic population-based national registers to characterize the genetic basis of autism and familial recurrence of autism.
Sven Sandin
Associate professor of psychiatry
Icahn School of Medicine at Mount Sinai
From this contributor
Autism is more heritable in boys than in girls
If boys have greater inherited liability for autism, the female protective effect may not fully explain the sex difference in prevalence.
Autism is more heritable in boys than in girls
Explore more from The Transmitter
Dose, scan, repeat: Tracking the neurological effects of oral contraceptives
We know little about how the brain responds to oral contraceptives, despite their widespread use. I am committed to changing that: I scanned my brain 75 times over the course of a year and plan to make my data openly available.
Dose, scan, repeat: Tracking the neurological effects of oral contraceptives
We know little about how the brain responds to oral contraceptives, despite their widespread use. I am committed to changing that: I scanned my brain 75 times over the course of a year and plan to make my data openly available.
Cracking the code of the extracellular matrix
Despite evidence for a role in plasticity and other crucial functions, many neuroscientists still view these proteins as “brain goop.” The field needs technical advances and a shift in scientific thinking to move beyond this outdated perspective.
Cracking the code of the extracellular matrix
Despite evidence for a role in plasticity and other crucial functions, many neuroscientists still view these proteins as “brain goop.” The field needs technical advances and a shift in scientific thinking to move beyond this outdated perspective.
Huntington’s disease gene variants past a certain size poison select cells
The findings—providing “the next step in the whole pathway”—help explain the disease’s late onset and offer hope that it has an extended therapeutic window.
Huntington’s disease gene variants past a certain size poison select cells
The findings—providing “the next step in the whole pathway”—help explain the disease’s late onset and offer hope that it has an extended therapeutic window.