Lydia Hickman is a graduate student in the Cook Lab at the University of Birmingham in the United Kingdom. In her Ph.D. work, she explores the fundamental biological mechanisms underlying motor function and social cognition in the context of autism and Parkinson’s disease. Lydia co-founded the U21 Autism Research Network, an international collaboration among six research groups aiming to improve diversity and inclusion in autism research.
Lydia Hickman
Graduate student
University of Birmingham in the United Kingdom
From this contributor
Ways to make autism research more diverse and inclusive
Scientists must focus on the importance of representative study samples and of engaging with diverse autism community members.
Ways to make autism research more diverse and inclusive
Explore more from The Transmitter
Neuroscientists weigh in on White House proposal for ‘moonshot infrastructure’ for connectomics
A report from the Trump administration calls for a “new golden age” of science, in which the federal government supports mission-driven collaborations to deliver high returns on research investments.
Neuroscientists weigh in on White House proposal for ‘moonshot infrastructure’ for connectomics
A report from the Trump administration calls for a “new golden age” of science, in which the federal government supports mission-driven collaborations to deliver high returns on research investments.
In memoriam: Kevin Dunbar, who traced the neural basis of human reasoning
Using imaging technologies, he transformed our understanding of how creativity and scientific discovery arise in the brain.
In memoriam: Kevin Dunbar, who traced the neural basis of human reasoning
Using imaging technologies, he transformed our understanding of how creativity and scientific discovery arise in the brain.
Neurons engage in risky, DNA-breaking business during migration
The cells later repair the damage, which occurs mostly in transcriptionally silent parts of the genome.
Neurons engage in risky, DNA-breaking business during migration
The cells later repair the damage, which occurs mostly in transcriptionally silent parts of the genome.