I am a postdoctoral fellow in the lab of Arnold Kriegstein at the University of California, San Francisco (UCSF). I focus on applying single-cell genomics techniques to study the development of specific cell types of the human brain, as well as to understand how these cell types are affected in various diseases, especially autism. Before starting my work at UCSF, I did my PhD at the University of Miami focusing on genomic analysis of autism. I did my B.S. and MS at Moscow State University in my native Russia, where I worked on animal models of epilepsy and Alzheimer’s disease.
Dmitry Velmeshev
Postdoctoral Scholar
University of California, San Francisco
From this contributor
Single-cell analysis suggests brain signaling problems in autism
Recent advances in technology allow researchers to measure RNA that is contained within the nucleus of a single brain cell.
![Neurons firing and lighting up](https://www.thetransmitter.org/wp-content/uploads/2019/05/20190522-NeuronsConvo-844.jpg)
Single-cell analysis suggests brain signaling problems in autism
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Cell population in brainstem coordinates cough, new study shows
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![](https://www.thetransmitter.org/wp-content/uploads/2024/09/1200_coughingmice-1024x720.webp)
Cell population in brainstem coordinates cough, new study shows
The work also adds to a growing body of evidence showing that mice, and their genetic toolbox, can be used to study cough.
In updated U.S. autism bill, Congress calls for funding boost, expanded scope
The current Autism CARES Act sunsets in late September.
![Capitol building](https://www.thetransmitter.org/wp-content/uploads/2024/09/capitolhill-1200-1024x692.webp)
In updated U.S. autism bill, Congress calls for funding boost, expanded scope
The current Autism CARES Act sunsets in late September.
Ketamine targets lateral habenula, setting off cascade of antidepressant effects
The drug’s affinity for overactive cells in the “anti-reward” region may help explain its rapid and long-lasting results.
![Illustration of ketamine blocking open ion channels in active NMDA receptors, quieting the cells and disrupting downstream signaling involved in depression.](https://www.thetransmitter.org/wp-content/uploads/2024/09/1200-ketamine-receptor-transmitter-neuroscience-1024x683.webp)
Ketamine targets lateral habenula, setting off cascade of antidepressant effects
The drug’s affinity for overactive cells in the “anti-reward” region may help explain its rapid and long-lasting results.