Charles A. Nelson is professor of pediatrics and neuroscience at Harvard Medical School and director of research at Boston Children’s Hospital’s Developmental Medicine Center.
Charles A. Nelson
Research director
Boston Children's Hospital
From this contributor
How separating children from parents causes irreparable harm
Science teaches us that housing children in institution-like settings is likely to cause severe and permanent damage to their minds and bodies.
How separating children from parents causes irreparable harm
Romanian orphans reveal clues to origins of autism
Understanding autism features in children who were deprived of social contact as infants could offer clues to the condition.
Romanian orphans reveal clues to origins of autism
Explore more from The Transmitter
Infant Brain Imaging Study findings, and more
Here is a roundup of autism-related news and research spotted around the web for the week of 23 March.
Infant Brain Imaging Study findings, and more
Here is a roundup of autism-related news and research spotted around the web for the week of 23 March.
Trading places: What happens when neuroscience turns into machine learning, and machine learning turns into neuroscience?
Neuroscience has become increasingly concerned with prediction, and machine learning with causal explanation, with each field adopting methods from the other. I asked eight experts to weigh in on what we stand to learn from this exchange.
Trading places: What happens when neuroscience turns into machine learning, and machine learning turns into neuroscience?
Neuroscience has become increasingly concerned with prediction, and machine learning with causal explanation, with each field adopting methods from the other. I asked eight experts to weigh in on what we stand to learn from this exchange.
Exon-skipping approach boosts levels of key Rett syndrome protein
Deleting a small region of the MECP2 gene partially restored function in neurons derived from people with Rett-associated variants.
Exon-skipping approach boosts levels of key Rett syndrome protein
Deleting a small region of the MECP2 gene partially restored function in neurons derived from people with Rett-associated variants.