Bahar Gholipour is a freelance science journalist based in New York. She focuses on the brain, neuroscience and psychology, and is passionate about reporting on artificial intelligence, genetics and the politics of healthcare. Her writing has appeared in various print and online publications including WIRED, New York Magazine and Scientific American. Bahar has a Masters degree in neuroscience from Ecole Normale Superieure in Paris, and has done graduate-level work at Stony Brook University’s School of Journalism.
Bahar Gholipour
Contributing writer
The Transmitter
From this contributor
In memoriam: Yves Frégnac, influential and visionary French neuroscientist
Frégnac, who died on 18 October at the age of 73, built his career by meeting neuroscience’s complexity straight on.
In memoriam: Yves Frégnac, influential and visionary French neuroscientist
By
Bahar Gholipour
18 December 2024 | 9 min read
Mistaking a duck for a skvader: How a conceptual form of circular analysis may taint many neuroscience studies
These logical loops are harder to spot than circularity involving noise in the data, but they result from neglecting something closer to home: existing knowledge about the brain.
Mistaking a duck for a skvader: How a conceptual form of circular analysis may taint many neuroscience studies
By
Bahar Gholipour
22 December 2023 | 7 min read
Double immune hit challenges female mice’s resistance to autism
Female mice can withstand a maternal antibody that triggers brain abnormalities and autism-like behaviors in males. But additional immune challenges spell trouble.
Double immune hit challenges female mice’s resistance to autism
By
Bahar Gholipour
23 October 2019 | 3 min read
New technique records neuronal teamwork in live mice
A new imaging technique detects specific groups of firing neurons in the brains of moving mice, enabling scientists to study brain activity linked to a particular behavior.
New technique records neuronal teamwork in live mice
By
Bahar Gholipour
23 October 2019 | 2 min read
Leaky mitochondria may play central role in fragile X syndrome
Some traits of fragile X syndrome may be due to problems with mitochondria, the cell’s energy factories.
Leaky mitochondria may play central role in fragile X syndrome
By
Bahar Gholipour
23 October 2019 | 2 min read
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Coding error caused layoffs at National Institute of Neurological Disorders and Stroke this week, source says
Thirty employees—including 11 lab heads—at the institute should “immediately return to work,” according to an email the institute’s Office of Human Resources sent to top administration at the institute Wednesday evening.
Coding error caused layoffs at National Institute of Neurological Disorders and Stroke this week, source says
Thirty employees—including 11 lab heads—at the institute should “immediately return to work,” according to an email the institute’s Office of Human Resources sent to top administration at the institute Wednesday evening.
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Sydney Wyatt
3 April 2025 | 3 min read
PTEN problems underscore autism connection to excess brain fluid
Damaging variants in the autism-linked gene cause congenital hydrocephalus—a buildup of cerebrospinal fluid in the brain—by turbocharging a downstream signaling pathway that promotes the growth of cells, according to a new study.
PTEN problems underscore autism connection to excess brain fluid
Damaging variants in the autism-linked gene cause congenital hydrocephalus—a buildup of cerebrospinal fluid in the brain—by turbocharging a downstream signaling pathway that promotes the growth of cells, according to a new study.
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Holly Barker
3 April 2025 | 4 min read
U.S. health agency purge includes 10 lab heads at National Institute of Neurological Disorders and Stroke
The reasons for selecting these researchers—who have led work on neuronal migration, dopamine receptors in neuronal signaling and the structure of ion channels, among other areas—remain unclear.
U.S. health agency purge includes 10 lab heads at National Institute of Neurological Disorders and Stroke
The reasons for selecting these researchers—who have led work on neuronal migration, dopamine receptors in neuronal signaling and the structure of ion channels, among other areas—remain unclear.
By
Angie Voyles Askham, Sydney Wyatt
2 April 2025 | 5 min read